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Identifying Genetic Causes of Hybrid Necrosis in Arrabidopsis lyrata
Umeå University, Faculty of Science and Technology, Department of Ecology and Environmental Sciences.
2019 (English)Independent thesis Basic level (degree of Bachelor), 10 credits / 15 HE creditsStudent thesis
Abstract [en]

Deleterious gene interactions due to accumulation of individual genetic variations between different lineages are a cause of population diversification by creating reproductive barriers that ultimately lead to differentiation of species. One type of deleterious interactions is called “hybrid necrosis”, in which epistatic interactions between some plant immunity genes (usually very variable) cause autoimmunities that produce a necrotic and dwarf phenotype. Hybrid necrosis has been widely studied in several plant species, such as Arabidopsis thaliana and many gene interactions were found for that plant. This study tests the applicability of these results on a close relative, A. lyrata, by crossing individuals from different populations and genotyping F2 progeny with polymorphic markers close to homologous sequences to those involved in hybrid necrosis in A. thaliana. Results suggest the possibility of a homologous gene to DM8 or DM9 in chromosome 7 to be involved in formation of hybrid necrosis.

Place, publisher, year, edition, pages
2019. , p. 41
Keywords [en]
ACD6, deleterious gene interactions, DM9, dwarfism, microsatellites
National Category
Evolutionary Biology
Identifiers
URN: urn:nbn:se:umu:diva-163444OAI: oai:DiVA.org:umu-163444DiVA, id: diva2:1352827
Presentation
2019-09-18, 10:00 (English)
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Examiners
Available from: 2019-09-23 Created: 2019-09-19 Last updated: 2019-09-23Bibliographically approved

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Valbuena-Gonzalo, Carlos
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CiteExportLink to record
Permanent link

Direct link
Cite
Citation style
  • apa
  • ieee
  • modern-language-association-8th-edition
  • vancouver
  • Other style
More styles
Language
  • de-DE
  • en-GB
  • en-US
  • fi-FI
  • nn-NO
  • nn-NB
  • sv-SE
  • Other locale
More languages
Output format
  • html
  • text
  • asciidoc
  • rtf