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Bidirectional signaling between Yersinia and its target cell.
Umeå University, Faculty of Medicine, Department of Molecular Biology (Faculty of Medicine).
Umeå University, Faculty of Medicine, Molecular Biology.
Umeå University, Faculty of Medicine, Molecular Biology.
Umeå University, Faculty of Medicine, Molecular Biology.
1998 (English)In: Folia microbiologica (Prague), ISSN 0015-5632, E-ISSN 1874-9356, Vol. 43, no 3, 263-273 p.Article in journal (Refereed) Published
Abstract [en]

Preventing the early host immune defense allows pathogenic Yersinia to proliferate in lymphatic tissue. This ability depends on signaling that occurs between the bacteria and the host cells. Following intimate contact with the target cell a signal is generated within the bacterium that results in increased expression of virulence-associated proteins that are subsequently delivered into the infected cell. These proteins, designated Yops, interfere with the host-cell signaling pathways that are normally activated to eliminate infectious agents.

Place, publisher, year, edition, pages
1998. Vol. 43, no 3, 263-273 p.
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Cell and Molecular Biology
Identifiers
URN: urn:nbn:se:umu:diva-33370DOI: 10.1007/BF02818611PubMedID: 9717253OAI: oai:DiVA.org:umu-33370DiVA: diva2:311640
Available from: 2010-04-22 Created: 2010-04-22 Last updated: 2017-12-12Bibliographically approved

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Fällman, MPersson, CSchesser, KWolf-Watz, Hans
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