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Invasin of Yersinia pseudotuberculosis activates human peripheral B cells.
Umeå University, Faculty of Medicine, Department of Molecular Biology (Faculty of Medicine).
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1996 (English)In: Infection and Immunity, ISSN 0019-9567, E-ISSN 1098-5522, Vol. 64, no 3, 829-35 p.Article in journal (Refereed) Published
Abstract [en]

The Yersinia pseudotuberculosis cell surface-located protein invasin was found to promote binding between the pathogen and resting peripheral B cells via beta 1 integrin receptors (CD29). B cells responded by expressing several activation markers and by growing, In contrast, T cells did not react, although these cells express CD29. An isogenic invA mutant failed to activate B cells. The mutation could be complemented by providing the invA+ gene in trans. Purified invasin alone did not activate B cells, although it was able to block the binding of bacteria to the cells.

Place, publisher, year, edition, pages
1996. Vol. 64, no 3, 829-35 p.
National Category
Medical and Health Sciences
Identifiers
URN: urn:nbn:se:umu:diva-61472PubMedID: 8641788OAI: oai:DiVA.org:umu-61472DiVA: diva2:567997
Available from: 2012-11-15 Created: 2012-11-15 Last updated: 2017-12-07
In thesis
1. Interactions between Yersinia pseudotuberculosis and host cells - role of invasin and YopH
Open this publication in new window or tab >>Interactions between Yersinia pseudotuberculosis and host cells - role of invasin and YopH
1999 (English)Doctoral thesis, comprehensive summary (Other academic)
Place, publisher, year, edition, pages
Umeå: Umeå universitet, 1999. 72 p.
Series
Umeå University medical dissertations, ISSN 0346-6612 ; 634
National Category
Medical and Health Sciences
Identifiers
urn:nbn:se:umu:diva-65079 (URN)91-7191-732-2 (ISBN)
Public defence
Föreläsningssalen, Institutionen för mikrobiologi, Umeå universitet, Umeå (English)
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retroaktiv registrering

Available from: 2013-02-06 Created: 2013-02-05 Last updated: 2013-02-06Bibliographically approved

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