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Wegmann, B., Lundquist, A., Eklund, A. & Villani, M. (2024). Bayesian modelling of effective and functional brain connectivity using hierarchical vector autoregressions. The Journal of the Royal Statistical Society, Series C: Applied Statistics
Öppna denna publikation i ny flik eller fönster >>Bayesian modelling of effective and functional brain connectivity using hierarchical vector autoregressions
2024 (Engelska)Ingår i: The Journal of the Royal Statistical Society, Series C: Applied Statistics, ISSN 0035-9254, E-ISSN 1467-9876Artikel i tidskrift (Refereegranskat) Epub ahead of print
Abstract [en]

Analysis of brain connectivity is important for understanding how information is processed by the brain. We propose a novel Bayesian vector autoregression hierarchical model for analysing brain connectivity within resting-state functional magnetic resonance imaging, and apply it to simulated data and a real data set with subjects in different groups. Our approach models functional and effective connectivity simultaneously and allows for both group- and single-subject inference. We combine analytical marginalization with Hamiltonian Monte Carlo to obtain highly efficient posterior sampling. We show that our model gives similar inference for effective connectivity compared to models with a common covariance matrix to all subjects, but more accurate inference for functional connectivity between regions compared to models with more restrictive covariance structures. A Stan implementation of our model is available on GitHub.

Ort, förlag, år, upplaga, sidor
Oxford University Press, 2024
Nyckelord
Bayesian inference, effective connectivity, functional connectivity, Hamiltonian Monte Carlo, hierarchical modelling, resting-state fMRI
Nationell ämneskategori
Sannolikhetsteori och statistik
Forskningsämne
statistik
Identifikatorer
urn:nbn:se:umu:diva-222725 (URN)10.1093/jrsssc/qlae014 (DOI)001186104900001 ()
Forskningsfinansiär
Riksbankens Jubileumsfond, P16-028:1Vetenskapsrådet, 2020-02846
Tillgänglig från: 2024-03-26 Skapad: 2024-03-26 Senast uppdaterad: 2024-03-26
Ljung, L., Jönsson, E., Franklin, J., Berglin, E., Lundquist, A. & Rantapää-Dahlqvist, S. (2024). Incidence and predisposing factors of extra-articular manifestations in contemporary rheumatoid arthritis. European journal of internal medicine
Öppna denna publikation i ny flik eller fönster >>Incidence and predisposing factors of extra-articular manifestations in contemporary rheumatoid arthritis
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2024 (Engelska)Ingår i: European journal of internal medicine, ISSN 0953-6205, E-ISSN 1879-0828Artikel i tidskrift (Refereegranskat) In press
Abstract [en]

Objective: Rheumatoid arthritis [RA) is a chronic inflammatory disease, with potential for extra-articular manifestations (ExRA). The incidence and predisposing factors for ExRA and the mortality were evaluated in an early RA inception cohort.

Methods: Patients (n = 1468; 69 % females, mean age (SD) 57.3(16.3) years) were consecutively included at the date of diagnosis, between 1 January 1996 and 31 December 2016, and assessed prospectively. In December 2016 development of ExRA was evaluated by a patient questionnaire and a review of medical records. Cumulative incidence and incidence rates were compared between 5-year periods and between patients included before and after 1 January 2001. Cox proportional hazard regression models were used to identify predictors for ExRA, and models with ExRA as time-dependent variables to estimate the mortality.

Results: After a mean (SD) follow-up of 9.3(4.9) years, 238 cases (23.3 %) had ExRA and 151 (14.7 %) had ExRA without rheumatoid nodules. Most ExRA developed within 5 years from diagnosis. Rheumatoid nodules (10.5 %) and keratoconjunctivitis sicca (7.1 %) were the most frequent manifestations, followed by pulmonary fibrosis (6.1 %). The ExRA incidence among more recently diagnosed patients was similar as to the incidence among patients diagnosed before 2001. Seropositivity, smoking and early biological treatment were associated with development of ExRA. After 15 years 20 % had experienced ExRA. ExRA was associated with increased mortality, HR 3.029 (95 % CI 2.177–4.213).

Conclusions: Early development of ExRA is frequent, particularly rheumatoid nodules. Predisposing factors were age, RF positivity, smoking and early biological treatment. The patients with ExRA had a 3-fold increase in mortality.

Nyckelord
ACPA, Disease modifying anti-rheumatic drugs, RF, Rheumatoid arthritis
Nationell ämneskategori
Reumatologi och inflammation
Identifikatorer
urn:nbn:se:umu:diva-224250 (URN)10.1016/j.ejim.2024.04.026 (DOI)2-s2.0-85192184871 (Scopus ID)
Forskningsfinansiär
Vetenskapsrådet, K2013–52X-20307–07–3Vetenskapsrådet, 2018–02551Stiftelsen Konung Gustaf V:s 80-årsfondReumatikerförbundetUmeå universitet
Tillgänglig från: 2024-05-15 Skapad: 2024-05-15 Senast uppdaterad: 2024-05-15
Erlandsson, A., Lundquist, A. & Olofsson, K. (2024). Mapping quality of life after balloon dilatation in subglottic stenosis using Dyspnea index and Short Form Health Survey-36. European Archives of Oto-Rhino-Laryngology
Öppna denna publikation i ny flik eller fönster >>Mapping quality of life after balloon dilatation in subglottic stenosis using Dyspnea index and Short Form Health Survey-36
2024 (Engelska)Ingår i: European Archives of Oto-Rhino-Laryngology, ISSN 0937-4477, E-ISSN 1434-4726Artikel i tidskrift (Refereegranskat) Epub ahead of print
Abstract [en]

Purpose: An accurate diagnosis and proper treatment plan are required to restore an adequate patent airway in fibrotic subglottic stenosis (SGS). Currently, the definitive treatment entails single-stage balloon dilatation with steroid injections. The primary aim was to evaluate successful airway restoration and general quality of life in cases with SGS in northern Sweden using robust patient reported outcomes.

Methods: All participants with need of surgical treatment due to SGS that had been referred to the department of otorhinolaryngology, University Hospital of Umeå from September 2020 to August 2023 was included. Exclusion criteria included malignant, extrathoracic or cartilaginous cause, age < 18 years, or incompetent to sign consent documents. We assessed the patient-reported outcome measures pre- as well as 3 months postoperatively.

Results: Of the 40 cases fulfilling the eligibility criteria’s, 33 cases completed the Dyspnea index (DI) and the short form health survey (SF-36) pre- as well as 3 months post-operatively. Receiver operating characteristics showed significant improvement in DI as well as in SF 36 scores post-operatively.

Conclusions: Evaluation of balloon dilatation in SGS in this cohort follow-up analysis shows clear improvement in patient quality of life using robust PROM 3 months postoperatively, ensuring the use of a safe and well-tolerated procedure.

Ort, förlag, år, upplaga, sidor
Springer Nature, 2024
Nyckelord
Balloon dilatation, Dyspnea index, Short form healthy survey, Subglottic stenosis
Nationell ämneskategori
Oto-rino-laryngologi
Identifikatorer
urn:nbn:se:umu:diva-224264 (URN)10.1007/s00405-024-08667-0 (DOI)001214210400002 ()38705896 (PubMedID)2-s2.0-85192082165 (Scopus ID)
Forskningsfinansiär
Cancerforskningsfonden i Norrland, AMP-19-973Cancerforskningsfonden i Norrland, AMP-21 1033Region Västerbotten, 967352Region Västerbotten, 979607
Tillgänglig från: 2024-05-14 Skapad: 2024-05-14 Senast uppdaterad: 2024-05-14
Lundquist, A., Lindberg, A., Eriksson Ström, J., Blomberg, A. & Backman, H. (2024). Number of follow-up years needed to identify a rapid decline in FEV1 [Letter to the editor]. American Journal of Respiratory and Critical Care Medicine, 209(1), 119-120
Öppna denna publikation i ny flik eller fönster >>Number of follow-up years needed to identify a rapid decline in FEV1
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2024 (Engelska)Ingår i: American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, E-ISSN 1535-4970, Vol. 209, nr 1, s. 119-120Artikel i tidskrift, Letter (Övrigt vetenskapligt) Published
Ort, förlag, år, upplaga, sidor
American Thoracic Society, 2024
Nationell ämneskategori
Lungmedicin och allergi
Forskningsämne
lungmedicin; epidemiologi
Identifikatorer
urn:nbn:se:umu:diva-216385 (URN)10.1164/rccm.202309-1664LE (DOI)37879065 (PubMedID)2-s2.0-85181395898 (Scopus ID)
Forskningsfinansiär
Hjärt-LungfondenVisare NorrRegion NorrbottenUmeå universitet
Tillgänglig från: 2023-11-09 Skapad: 2023-11-09 Senast uppdaterad: 2024-01-11Bibliografiskt granskad
Nyberg, L., Andersson, M., Lundquist, A., Baaré, W. F. .., Bartrés-Faz, D., Bertram, L., . . . Walhovd, K. B. (2023). Individual differences in brain aging: heterogeneity in cortico-hippocampal but not caudate atrophy rates. Cerebral Cortex, 33(9), 5075-5081
Öppna denna publikation i ny flik eller fönster >>Individual differences in brain aging: heterogeneity in cortico-hippocampal but not caudate atrophy rates
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2023 (Engelska)Ingår i: Cerebral Cortex, ISSN 1047-3211, E-ISSN 1460-2199, Vol. 33, nr 9, s. 5075-5081Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

It is well documented that some brain regions, such as association cortices, caudate, and hippocampus, are particularly prone to age-related atrophy, but it has been hypothesized that there are individual differences in atrophy profiles. Here, we document heterogeneity in regional-atrophy patterns using latent-profile analysis of 1,482 longitudinal magnetic resonance imaging observations. The results supported a 2-group solution reflecting differences in atrophy rates in cortical regions and hippocampus along with comparable caudate atrophy. The higher-atrophy group had the most marked atrophy in hippocampus and also lower episodic memory, and their normal caudate atrophy rate was accompanied by larger baseline volumes. Our findings support and refine models of heterogeneity in brain aging and suggest distinct mechanisms of atrophy in striatal versus hippocampal-cortical systems.

Ort, förlag, år, upplaga, sidor
Oxford University Press, 2023
Nyckelord
aging, individual differences, caudate, hippocampus, cortex
Nationell ämneskategori
Neurovetenskaper
Forskningsämne
medicin
Identifikatorer
urn:nbn:se:umu:diva-201287 (URN)10.1093/cercor/bhac400 (DOI)000863898100001 ()36197324 (PubMedID)2-s2.0-85159256770 (Scopus ID)
Forskningsfinansiär
Knut och Alice Wallenbergs StiftelseEU, Horisont 2020, 732592
Tillgänglig från: 2022-11-27 Skapad: 2022-11-27 Senast uppdaterad: 2023-06-07Bibliografiskt granskad
Nyberg, L., Andersson, M. & Lundquist, A. (2023). Longitudinal change-change associations of cognition with cortical thickness and surface area. Aging Brain, 3, Article ID 100070.
Öppna denna publikation i ny flik eller fönster >>Longitudinal change-change associations of cognition with cortical thickness and surface area
2023 (Engelska)Ingår i: Aging Brain, E-ISSN 2589-9589, Vol. 3, artikel-id 100070Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

Age-related changes in cortical volumes are well established but relatively few studies probed its constituents, surface area (SA) and thickness (TH). Here we analyzed 10-year, 3-waves longitudinal data from a large sample of healthy individuals (baseline age = 55- 80). The findings showed marked age-related changes of SA in frontal, temporal, and parietal association cortices, and Bivariate Latent Change Score models revealed significant SAassociations with changes in speed of processing in both the 5- and 10-year models. The corresponding results for TH revealed a late onset of thinning and significant associations with reduced cognition in the 10-year model only. Taken together, our findings suggest that cortical surface area shrinks and impacts information-processing capacity gradually in aging, whereas cortical thinning only manifests and impacts fluid cognition in advanced aging. 

Ort, förlag, år, upplaga, sidor
Elsevier, 2023
Nyckelord
Cortex, Thickness, Surface area, Cognition, Speed
Nationell ämneskategori
Neurovetenskaper Neurologi
Identifikatorer
urn:nbn:se:umu:diva-222727 (URN)10.1016/j.nbas.2023.100070 (DOI)001133947400001 ()37408792 (PubMedID)
Forskningsfinansiär
Knut och Alice Wallenbergs StiftelseVetenskapsrådet, 2018-05973
Tillgänglig från: 2024-03-26 Skapad: 2024-03-26 Senast uppdaterad: 2024-03-27Bibliografiskt granskad
Backman, H., Blomberg, A., Lundquist, A., Strandkvist, V., Sawalha, S., Nilsson, U., . . . Lindberg, A. (2023). Lung function trajectories and associated mortality among adults with and without airway obstruction. American Journal of Respiratory and Critical Care Medicine, 208(10), 1063-1074
Öppna denna publikation i ny flik eller fönster >>Lung function trajectories and associated mortality among adults with and without airway obstruction
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2023 (Engelska)Ingår i: American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, E-ISSN 1535-4970, Vol. 208, nr 10, s. 1063-1074Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

Rationale: Spirometry is essential for diagnosis and assessment of prognosis in COPD.

Objectives: To identify FEV1 trajectories and their determinants, based on annual spirometry measurements among individuals with and without airway obstruction. Furthermore, to assess mortality in relation to trajectories.

Methods: In 2002-04, individuals with airway obstruction (AO) (FEV1/VC<0.70, n=993) and age- and sex-matched non-obstructive (NO) referents were recruited from population-based cohorts. Annual spirometries until 2014 were utilized in joint-survival Latent Class Mixed Models to identify lung function trajectories. Mortality data were collected during 15 years of follow-up.

Results: Three trajectories were identified among the AO-cases and two among the NO referents. Trajectory membership was driven by baseline FEV1%predicted (%pred) in both groups and additionaly, pack-years in AO and current smoking in NO. Longitudinal FEV1%pred level depended on baseline FEV1%pred, pack-years and obesity. The trajectories were distributed: 79.6% T1AO FEV1-high with normal decline, 12.8% T2AO FEV1-high with rapid decline, and 7.7% T3AO FEV1-low with normal decline (mean 27, 72 and 26 mL/year) among AO-individuals, and 96.7% T1NO FEV1-high with normal decline and 3.3% T2NO FEV1-high with rapid decline (mean 34 and 173 mL/year) among referents. Hazard for death was increased for T2AO (HR1.56) and T3AO (HR3.45) vs. T1AO, and for T2NO (HR2.99) vs. T1NO.

Conclusions: Three different FEV1 trajectories were identified among those with airway obstruction and two among the referents, with different outcomes in terms of FEV1-decline and mortality. The FEV1 trajectories among airway obstructive and the relationship between low FVC and trajectory outcome are of particular clinical interest.

Ort, förlag, år, upplaga, sidor
American Thoracic Society, 2023
Nyckelord
prognosis, chronic obstructive pulmonary disease, FEV1, natural history
Nationell ämneskategori
Lungmedicin och allergi
Forskningsämne
lungmedicin; epidemiologi
Identifikatorer
urn:nbn:se:umu:diva-212976 (URN)10.1164/rccm.202211-2166oc (DOI)001099650500015 ()37460250 (PubMedID)2-s2.0-85183557430 (Scopus ID)
Forskningsfinansiär
Hjärt-LungfondenUmeå universitetVisare NorrRegion NorrbottenSvensk Lungmedicinsk FöreningRegion Västerbotten
Tillgänglig från: 2023-08-17 Skapad: 2023-08-17 Senast uppdaterad: 2024-04-08Bibliografiskt granskad
Brink, M., Berglin, E., Mohammad, A. J., Lundquist, A., Gjertsson, I., Alexeyenko, A., . . . Rantapää-Dahlqvist, S. (2023). Protein profiling in presymptomatic individuals separates myeloperoxidase-antineutrophil cytoplasmic antibody and proteinase 3-antineutrophil cytoplasmic antibody vasculitides. Arthritis & Rheumatology, 75(6), 996-1006
Öppna denna publikation i ny flik eller fönster >>Protein profiling in presymptomatic individuals separates myeloperoxidase-antineutrophil cytoplasmic antibody and proteinase 3-antineutrophil cytoplasmic antibody vasculitides
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2023 (Engelska)Ingår i: Arthritis & Rheumatology, ISSN 2326-5191, E-ISSN 2326-5205, Vol. 75, nr 6, s. 996-1006Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

Objective: Antineutrophil cytoplasmic antibody (ANCA)–associated vasculitis (AAV) is a chronic relapsing condition with unknown etiology. To gain insight into the molecular processes underlying the disease, we examined biomarkers in blood samples collected prior to symptom onset.

Methods: The National Patient Register and Cause of Death register were searched for AAV-related International Classification of Diseases, Ninth Revision and Tenth Revision codes and linked to the registers from 5 biobanks. Eighty-five AAV patients with samples predating symptom onset of AAV were identified. For each case of AAV, 2 matched controls were included. Proteinase 3 (PR3)–ANCA and myeloperoxidase (MPO)–ANCA expression levels were analyzed using enzyme-linked immunosorbent assays. Using an Olink Inflammation panel, 73 of 92 proteins were included after quality control. Data were replicated in a second cohort of 48 presymptomatic individuals and 96 controls.

Results: Of the 20 proteins with the lowest P values in the original cohort, 7 were replicated in the second cohort and 5 proteins were found to be significant between the groups in a meta-analysis. Eleven different pathways were identified in network enrichment analyses and were found to be significant in both cohorts. Stratification of samples obtained ≤5 years before symptom onset showed significant levels of CCL23, vascular endothelial growth factor A, and hepatocyte growth factor, which were also increased at borderline significant levels in the replication cohort (interleukin-6 was found to be significantly increased in the replication cohort). In presymptomatic AAV patients, 6 proteins were associated with MPO-ANCA positivity, and 7 proteins were associated with PR3-ANCA positivity.

Conclusion: To our knowledge, this is the first study to identify protein markers preceding symptom onset in AAV patients. These findings set the stage for further research into the underlying cellular and molecular mechanisms in the pathogenesis of AAV and the diversification of patients into PR3-ANCA+ and MPO-ANCA+ subphenotypes. (Figure presented.).

Nationell ämneskategori
Reumatologi och inflammation
Identifikatorer
urn:nbn:se:umu:diva-211991 (URN)10.1002/art.42425 (DOI)000967341700001 ()36533851 (PubMedID)2-s2.0-85147033853 (Scopus ID)
Forskningsfinansiär
Vetenskapsrådet, 2018-02551Stiftelsen Konung Gustaf V:s 80-årsfondUmeå universitetRegion Västerbotten
Tillgänglig från: 2023-07-12 Skapad: 2023-07-12 Senast uppdaterad: 2023-07-12Bibliografiskt granskad
Vikner, T., Karalija, N., Eklund, A., Malm, J., Lundquist, A., Gallewicz, N., . . . Wåhlin, A. (2022). 5-year associations among cerebral arterial pulsatility, perivascular space dilation, and white matter lesions. Annals of Neurology, 92(5), 871-881
Öppna denna publikation i ny flik eller fönster >>5-year associations among cerebral arterial pulsatility, perivascular space dilation, and white matter lesions
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2022 (Engelska)Ingår i: Annals of Neurology, ISSN 0364-5134, E-ISSN 1531-8249, Vol. 92, nr 5, s. 871-881Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

Objective: High cerebral arterial pulsatility index (PI), white matter lesions (WMLs), enlarged perivascular spaces (PVSs), and lacunar infarcts are common findings in the elderly population, and considered indicators of small vessel disease (SVD). Here, we investigate the potential temporal ordering among these variables, with emphasis on determining whether high PI is an early or delayed manifestation of SVD.

Methods: In a population-based cohort, 4D flow MRI data for cerebral arterial pulsatility was collected for 159 participants at baseline (age 64–68), and for 122 participants at follow-up 5 years later. Structural MRI was used for WML and PVS segmentation, and lacune identification. Linear mixed-effects (LME) models were used to model longitudinal changes testing for pairwise associations, and latent change score (LCS) models to model multiple relationships among variables simultaneously.

Results: Longitudinal 5-year increases were found for WML, PVS, and PI. Cerebral arterial PI at baseline did not predict changes in WML or PVS volume. However, WML and PVS volume at baseline predicted 5-year increases in PI. This was shown for PI increases in relation to baseline WML and PVS volumes using LME models (R (Formula presented.) 0.24; p < 0.02 and R (Formula presented.) 0.23; p < 0.03, respectively) and LCS models ((Formula presented.) = 0.28; p = 0.015 and (Formula presented.) = 0.28; p = 0.009, respectively). Lacunes at baseline were unrelated to PI.

Interpretation: In healthy older adults, indicators of SVD are related in a lead–lag fashion, in which the expression of WML and PVS precedes increases in cerebral arterial PI. Hence, we propose that elevated PI is a relatively late manifestation, rather than a risk factor, for cerebral SVD. 

Ort, förlag, år, upplaga, sidor
John Wiley & Sons, 2022
Nationell ämneskategori
Neurologi
Identifikatorer
urn:nbn:se:umu:diva-199208 (URN)10.1002/ana.26475 (DOI)000843724700001 ()36054261 (PubMedID)2-s2.0-85136905097 (Scopus ID)
Forskningsfinansiär
Stiftelsen för strategisk forskning (SSF)Region Västerbotten, 2017‐04949Knut och Alice Wallenbergs Stiftelse, 2017‐04949Max Planck GesellschaftVetenskapsrådet, 2017‐02217Vetenskapsrådet, 421‐2012‐648
Tillgänglig från: 2022-09-08 Skapad: 2022-09-08 Senast uppdaterad: 2023-05-04Bibliografiskt granskad
Salami, A., Adolfsson, R., Andersson, M., Blennow, K., Lundquist, A., Nordin Adolfsson, A., . . . Nyberg, L. (2022). Association of APOE ɛ4 and Plasma p-tau181 with Preclinical Alzheimer's Disease and Longitudinal Change in Hippocampus Function. Journal of Alzheimer's Disease, 85(3), 1309-1320
Öppna denna publikation i ny flik eller fönster >>Association of APOE ɛ4 and Plasma p-tau181 with Preclinical Alzheimer's Disease and Longitudinal Change in Hippocampus Function
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2022 (Engelska)Ingår i: Journal of Alzheimer's Disease, ISSN 1387-2877, E-ISSN 1875-8908, Vol. 85, nr 3, s. 1309-1320Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

BACKGROUND: The Apolipoprotein E (APOE) ɛ4 allele has been linked to increased tau phosphorylation and tangle formation. APOE ɛ4 carriers with elevated tau might be at the higher risk for Alzheimer's disease (AD) progression. Previous studies showed that tau pathology begins early in areas of the medial temporal lobe. Similarly, APOE ɛ4 carriers showed altered hippocampal functional integrity. However, it remains unknown whether the influence of elevated tau accumulation on hippocampal functional changes would be more pronounced for APOE ɛ4 carriers.

OBJECTIVE: We related ɛ4 carriage to levels of plasma phosphorylated tau (p-tau181) up to 15 years prior to AD onset. Furthermore, elevated p-tau181 was explored in relation to longitudinal changes in hippocampal function and connectivity.

METHODS: Plasma p-tau181 was analyzed in 142 clinically defined AD cases and 126 matched controls. The longitudinal analysis involved 87 non-demented individuals (from population-based study) with two waves of plasma samples and three waves of functional magnetic resonance imaging during rest and memory encoding.

RESULTS: Increased p-tau181 was observed for both ɛ4 carriers and non-carriers close to AD onset, but exclusively for ɛ4 carriers in the early preclinical groups (7- and 13-years pre-AD). In ɛ4 carriers, longitudinal p-tau181 increase was paralleled by elevated local hippocampal connectivity at rest and subsequent reduction of hippocampus encoding-related activity.

CONCLUSION: Our findings support an association of APOE ɛ4 and p-tau181 with preclinical AD and hippocampus functioning.

Ort, förlag, år, upplaga, sidor
IOS Press, 2022
Nyckelord
Alzheimer’s disease, APOE, fMRI, hippocampus, longitudinal, magnetic resonance imaging, p-tau181, phosphorylated tau, population-based
Nationell ämneskategori
Neurovetenskaper
Identifikatorer
urn:nbn:se:umu:diva-192513 (URN)10.3233/JAD-210673 (DOI)000752449800030 ()34924376 (PubMedID)2-s2.0-85124174700 (Scopus ID)
Tillgänglig från: 2022-02-22 Skapad: 2022-02-22 Senast uppdaterad: 2024-04-08Bibliografiskt granskad
Projekt
Metoder för longitudinella hjärnavbildningsstudier med icke-slumpmässigt bortfall. [P16-0628:1_RJ]; Umeå universitet
Organisationer
Identifikatorer
ORCID-id: ORCID iD iconorcid.org/0000-0003-1524-0851

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