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3D imaging of an entire pancreas shows inverse proportions of extra-islet versus islet-associated β cells in late-onset type 1 diabetes
Umeå University, Faculty of Medicine, Department of Medical and Translational Biology.
Umeå University, Faculty of Medicine, Department of Medical and Translational Biology.ORCID iD: 0000-0002-0712-8256
Umeå University, Faculty of Medicine, Department of Medical and Translational Biology.ORCID iD: 0000-0002-4252-6903
Umeå University, Faculty of Medicine, Department of Medical and Translational Biology.ORCID iD: 0000-0003-0232-1812
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2026 (English)In: Science Advances, E-ISSN 2375-2548, Vol. 12, no 21, article id eaed0496Article in journal (Refereed) Published
Abstract [en]

Residual β cell function can positively affect diabetes regulation in type 1 diabetes (T1D), but details on residual β cell mass distribution in T1D is largely lacking in a whole organ context. Implementing an optical 3D imaging pipeline, we generated a complete account of the remaining β cells throughout an entire human late onset T1D pancreas at a microscopic resolution. Our data show that most of the residual β cells were present as scattered individual cells or as punctated clusters of individual β cells, spatially separated from each other and all other endocrine cell types. Compared to islet-associated β cells, extra-islet β cells appeared in a substantially higher relative abundance in the head region of the T1D pancreas. This 3D depiction of an entire T1D pancreas shows that individual β cells may be preserved and/or formed in a highly regionalized manner, potentially reflecting key aspects of disease dynamics, advocating for increased focus on extra-islet islet β cells in attempts to develop strategies for pancreatic β cell preservation in T1D.

Place, publisher, year, edition, pages
American Association for the Advancement of Science (AAAS), 2026. Vol. 12, no 21, article id eaed0496
National Category
Cell and Molecular Biology Endocrinology and Diabetes
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URN: urn:nbn:se:umu:diva-254544DOI: 10.1126/sciadv.aed0496ISI: 001772851400025PubMedID: 42172326Scopus ID: 2-s2.0-105039958205OAI: oai:DiVA.org:umu-254544DiVA, id: diva2:2070694
Funder
The Kempe Foundations, JCSMK24-0063Swedish Research Council, 2023-02221Swedish Research Council, 2025-04958Umeå University, FS 2.1.6-2026-20Umeå University, FS 2.1.6-74-24Swedish Child Diabetes Foundation, 2024-0037-förebyggNovo Nordisk Foundation, NNF21OC0069771Novo Nordisk Foundation, 0084520Novo Nordisk Foundation, NNF24OC0092100Insamlingsstiftelsen Diabetes Wellness, PG21-6566Ernfors Foundation, 2023Nils Erik Holmstens forskningsstiftelse, 2023Diabetesfonden, DIA2024-914Available from: 2026-06-12 Created: 2026-06-12 Last updated: 2026-06-12Bibliographically approved

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Lehrstrand, JoakimHahn, MaxMorén, BjörnDavies, Wayne I. L.Alanentalo, TomasAhlgren, Ulf

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