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[Commentary] Chlamydia Anti-apoptosis - A By-product of Metabolic Reprogramming?
INSERM U1138, Centre de Recherche des Cordeliers, Paris, France; Equipe 11 labellisée Ligue Nationale contre le Cancer, Centre de Recherche des Cordeliers, Paris, France; Université Paris Descartes, Paris, France; Metabolomics and Cell Biology Platforms, Institut Gustave Roussy, Villejuif, France.ORCID iD: 0000-0002-5607-8902
2017 (English)In: EBioMedicine, E-ISSN 2352-3964, Vol. 23, p. 2-3Article in journal (Refereed) Published
Place, publisher, year, edition, pages
2017. Vol. 23, p. 2-3
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Microbiology in the medical area Cell and Molecular Biology
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URN: urn:nbn:se:umu:diva-181817DOI: 10.1016/j.ebiom.2017.08.018PubMedID: 28844411OAI: oai:DiVA.org:umu-181817DiVA, id: diva2:1540068
Note

Refers to: Munir A. Al-Zeer, Audrey Xavier, Mohammad Abu Lubad, Janine Sigulla, Mirjana Kessler, Robert Hurwitz, Thomas F. Meyer; Chlamydia trachomatis Prevents Apoptosis Via Activation of PDPK1-MYC and Enhanced Mitochondrial Binding of Hexokinase II, EBioMedicine, Volume 23, September 2017, Pages 100-110. DOI: 10.1016/j.ebiom.2017.08.005

Available from: 2021-03-26 Created: 2021-03-26 Last updated: 2021-03-26Bibliographically approved

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Sixt, Barbara Susanne

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